Xenoestrogens and Xenoandrogens

In Nature?
Despite efforts to use homosexuality in nature to argue that homosexuality is a natural phenomenon, in actuality homosexuality has been induced in nature and within humans specifically by scientists using artificial drugs which imitate male and female hormones, androgen and estrogen respectively. As such, the increasing prevalence of homosexuality appears attributable to scientists deliberately creating hormone-imitating chemicals to influence sexuality (particularly since many of the scientists studying homosexuality are themselves homosexual ), possibly in an attempt to slow the rate of global population growth.

Xenoestrogens and Xenoandrogens
Xenoestrogens and xenoandrogens are commonly used in foods, cosmetics, pesticides, and even baby toys. Over the past 70 years unnatural estrogen and androgen-imitating chemicals and pesticides have been introduced into nature through industrial, agricultural, and chemical companies which mimic male and female pheromones. Thus ironically, even if there is a genetic cause for homosexuality, it is an unnatural one caused by scientific tampering with hormones.

A xenoestrogen is a synthetic form of estrogen, and it is found in a lot more things than just plastic. Xenoestrogens are also found in pesticides, herbicides, fungicides, household chemicals, dry cleaning chemicals, body creams, shampoos, soaps, and even commercial meats that come from animals that are pumped full of estrogen-laden drugs. In other words, they are pretty much everywhere. The term xenoestrogen simply means 'foreign estrogen' or 'estrogen mimicker.' Once introduced in the body, xenoestrogens try to fool the body into thinking they are natural estrogens, but they are far from it. The accumulations of these xenoestrogens quickly create hormonal imbalances and endocrine system disruption. The end result can be felt all the way from mood swings, weight gain and depression to full blown cancer. In the last 70 years, xenoestrogens have not only found their way into products that we use every day, but alarmingly, traces of xenoestrogens are found in food containers or even food itself. This allows these synthetic compounds a direct path into our body via our unsuspecting mouths. -Brad King, TotalHealth Magazine

Per the following NCBI paper, scientists are able to control sexual urges in animals and humans largely at will through use of sex steroids that disrupt the endocrine system:

"Male-typical sexual orientation is controlled at least in part by the POA (like sexual behavior), and it differentiates under the influence of pre-/perinatal sex steroids. Many Sex Differences in Humans Are Organized by Embryonic Sex Steroids: Do these endocrine mechanisms demonstrated in animals have any significance in humans? The answer to this question should be considered in two steps. 1) Do we have any evidence that sex steroids are, in humans like in animals, implicated in the sexual differentiation of morphology (e.g. genital structures) but also of brain (e.g. SDN-POA) and sexual behavior? And 2) are there any data indicating that embryonic sex steroids have, like in animals, organizational effects on sexual orientation in humans? The answer to the first of these questions is clearly yes, and there is probably no need to elaborate on the arguments supporting this conclusion especially in an endocrine journal... A potential implication of the embryonic hormonal environment in the control of sexual orientation is also supported by studies of various clinical disorders that affect the endocrine system during fetal life. In some cases, these early endocrine disruptions lead to a complete sex reversal, so that, postnatally, subjects are raised assuming a sex (gender) that is opposite to their genetic sex." -Jacques Balthazart, "Minireview: Hormones and Human Sexual Orientation," Endocrinology 152(8): 2937–2947.

In 2007, the attempts by researchers to turn sheep gay ironically caused outrage from the LGBT, who thought the results could be used against their agenda. As such, the liberal Slate Magazine argued at the time that the researchers' actions were supportive of the LGBT.

"The sheep researchers—Charles Roselli of Oregon Health and Science University and Fred Stormshak of Oregon State University—are investigating biological factors in homosexuality... Nor has Roselli tried, in any experiment, to make sheep turn out straight. He has tried the opposite: to make them turn out gay. People for the Ethical Treatment of Animals accuses them of trying to “cure” homosexuality. Not true. Roselli and OHSU’s publicist, Jim Newman, have spent months dousing this fire. Roselli has never said or written anything against homosexuality. In fact, he has said that studies suggesting a biological basis for homosexuality tend to encourage tolerance... PETA thinks this study threatens to advance the eradication of homosexuality. But look more closely. The paper’s title is 'Influence of castration and estrogen replacement on sexual behavior of female-oriented, male-oriented, and asexual rams.' And that’s what the study examines: All the rams, not just the gay ones, are castrated and given estrogen. The point isn’t to make the gay rams straight; it’s to 'restore sexual behavior' in general." -William Saletan, Slate Magazine.

Unnaturally Altering Sexual Preferences and Genitals
Mankind's genetic tampering increasingly appears to be the reason for homosexuality in nature. Scientists deliberately manipulate newt pheromones to introduce homosexuality in nature, as one example. Xenoestrogens have been shown to produce homosexual urges in snakes by confusing genders through smell. Estrogen has also been shown to alter gender preference in quail and chicken as well. Artificial sex hormones have also been used to induce homosexuality in fruit flies, rats, hamsters, ferrets, pigs, zebra finches, and dogs:

"A new study finds that both drugs and genetic manipulation can turn the homosexual behavior of fruit flies on and off within a matter of hours. While the genetic finding supports the thinking that homosexuality is hard-wired, the drug finding surprisingly suggests it's not that simple. In fact, homosexuality in the fruit flies seems to be regulated by how they interpret the scent of another... In the new work, University of Illinois at Chicago researcher David Featherstone and coworkers discovered a gene in fruit flies they call 'genderblind,' or GB. A mutation in GB turns flies bisexual. GB transports the neurotransmitter glutamate to brain cells. Altering levels of glutamate change the strength of nerve cell junctions, called synapses, which play a key role in human and animal behavior. Post-doctoral researcher Yael Grosjean found that all male fruit flies with a mutation in their GB gene courted other males." -Robert Roy Britt, LiveScience.

"In rats, hamsters, ferrets, pigs, zebra finches, and possibly dogs, either early castration of males or early testosterone treatment of females or both have been shown to change or reverse sexual orientation." -Elizabeth Adkins Regan, "Sex Hormones and Sexual Orientation in Animals," Psychobiology 6(4): 335-347.

Scientists were able to produce homosexuality in nature in rodents and rhesus monkeys by exposing them to xenoandrogens and diethylstilbestrol. In the case of mice, they tampered with the genetic code in an attempt to produce homosexuality without the use of steroids.

"The reproducibility of this effect has been questioned, but if the effect of DES is real, which will be difficult to confirm given that this treatment has been abandoned for a long time, it would indicate that estrogens as well as androgens (testosterone) are able to masculinize sexual orientation. This would fit with rodent data, where many effects of testosterone on sexual differentiation are produced after conversion into estradiol by aromatase in the brain, but would be in conflict with other data from humans that assign a prominent role to androgens in sexual differentiation. Note, however, that in rhesus monkeys, fetal exposure to DES was shown to increase adult mounting behavior although not to the male-typical level." -Jacques Balthazart, "Minireview: Hormones and Human Sexual Orientation," Endocrinology 152(8): 2937–2947.

"These studies indicated that the type of sexual behaviour (male- or female-typical) displayed by an adult individual is determined by exposure to steroids during the early stages of life... researchers took advantage of a mutated mouse model in which the SRY gene was no longer functional, so that it could no longer induce the formation of testes (the XYSRY2 mouse). In another mouse line, they additionally translocated the SRY gene to an autosome, so that XX females would develop testes during the early embryonic life (XXSRY mouse)." Jacques Balthazart, "Sex Differences in Partner Preferences in Humans and Animals.", Phil. Trans. R. Soc. B 371:20150118.

Xenoestrogens and xenoandrogens have resulted in fish, amphibians, and aquatic reptiles becoming homosexual as well, including cases where they have intrasex genitalia.

"That synthetic chemicals in the environment can wreak havoc on the endocrine system of animals is irrefutable when considering aquatic species. Exposure from pesticide runoff, industrial waste, and oral contraceptives in effluent of water treatment plants are the primary sources, with fish, amphibians and aquatic reptiles literally swimming in the stuff, and illustrating the price with abnormal genitalia, increased incidences of intrasex individuals, and infertility." -Margaret M. McCarthy, Physiological Reviews 88:91-134,American Physiological Society

Xenoestrogens and xenoandrogens not only alter sexual preference, but can unnaturally alter genitals as well, producing male-like genitals in females and vice versa.

"These results may or may not apply to humans, of course, but some researchers have investigated human females who were 'androgenized' in the womb because of drugs given to their mothers to prevent miscarriage. By and large, the results of these studies are similar to the experimental studies-androgenized human females show similar patterns of higher aggression. Some scholars take these results to indicate that biological differences between males and females are responsible for the male-female difference in aggression; others suggest that even these results are not conclusive because females who get more androgen show generally disturbed metabolic systems, and general metabolic disturbance may itself increase aggressiveness. Furthermore, androgen-injected females may look more like males because they develop male-like genitals; therefore, they may be treated like males." -Carol R. Ember, Melvin Ember, and Peter N. Peregrine, Anthropology: Fourteenth Edition.

"Butyl benzyl phthalate (BBP) is another endocrine disruptor that has been reported to be an estrogenic compound and a partial agonist for ER (Jobling et al., 1995; Zacharewski et al., 1998; Andersen et al., 1999). It is used as a plasticizer, and is widely used in food wraps and cosmetic formulations. The International Program of Chemical Safety (IPCS) concluded that BBP exposure to the general population is based almost entirely on food intake, because the concentrations of BBP in air, drinking water, and soil are very low making intakes from these routes essentially negligible... When administered during sexual differentiation, BBP also causes male reproductive tract malformation of the external genitalia, sex accessory glands, epididymides and testes (Gray et al., 2000)." -S.V. Fernandez and J. Russo, "Estrogens and Xenoestrogens in Breast Cancer," Toxicol Pathol 38(1): 110–122.

How Scientists Created LGBT Urges in Humans: Diethylstilbestrol
The most prominent xenoestrogen, diethylstilbestrol (DES), was created in 1938 and from 1940-71 was given to as many as 5-10 million pregnant women in the United States by the USDA under the mistaken belief it would prevent miscarriages. It was also distributed in France, Spain, and the Netherlands. Studies have shown that those taking diethylstilbestrol have increased likelihood of homosexuality.

"Treatment of pregnant mothers with diethylstilbestrol (DES): Between 1939 and 1960, about 2 million pregnant women were treated with DES in Europe and the United States to prevent spontaneous miscarriages. This treatment turned out to be ineffective and also to have detrimental long-term consequences, but one of the unexpected outcomes was that girls born from these treated mothers showed a significant increase in nonheterosexual (bisexual or homosexual) fantasies or sexual activity, whereas the socialization of these subjects was fundamentally consistent with their genetic female sex." -Jacques Balthazart, "Minireview: Hormones and Human Sexual Orientation," Endocrinology 152(8): 2937–2947.

The FDA continued to approve diethylstilbestrol for estrogen-replacement therapy until the late 1980s. It is now known that DES causes serious health problems including breast cancer, vaginal and cervical cancer, clear cell cancer, testical cysts, premature births, infertility, and genital abnormalities.

The U.S. Military's 'Gay Bomb' Proposal
In 1994, the U.S. Air Force proposed use of a gay bomb against foreign combatants as a way to weaken opposing enemy forces through chemical hormones by making soldiers "sexually irresistible to one another." The project's originators even won a 2007 IG Nobel Award from Harvard University for the unorthodox proposal. As such, the possibility exists that either A) the U.S. government is targeting people with chemical warfare (e.g. DES) to make them homosexual, or that B) a foreign government (like China) is doing so.

"The unusual weapon, confirmed by Pentagon sources, is a 'gay bomb.' The project, which officials say has now been scrapped, was to come up with a device to release unspecified hormones that could be absorbed through the skin or lungs, thereby incapacitating soldiers who—according to the plan—would be too busy swooning over each other in homosexual ecstasy to waste any time dashing about planting roadside bombs." -BBC News

"The Pentagon maintains that the love affair with the gay bomb idea was brief. However, the Sunshine Project thinks the Pentagon doth protest too much, finding that they 'submitted the proposal to the highest scientific review body in the country for them to consider.' Indeed, the proposal’s information was submitted to the National Academy of Sciences in 2002... For the attempt at making a gay bomb, the Wright Lab had the honor of winning the Ig Nobel Peace Prize in 2007. As the prize is organized by the Annals of Improbable Research, it seems to be an excellent home for the project, though perhaps a step down from the National Academy of Sciences." -O'Rene D. Ashley, Gizmodo

Hormone Replacement Therapy
Xenoestrogens are used in Hormone Replacement Therapy (HRT) which is used to treat 'Gender Dysphoria' / 'Gender Identity Disorder.' In essence, estrogen-imitating chemicals are used to change the sexual characteristics of those who aren't happy with the gender they are born with. HRT has also been used extensively to treat menopause symptoms with Menopausal Hormone Therapy (MHT). Like their counterparts xenoestrogens, xenoandrogens (which imitate the male hormone, testosterone) likewise cause reproductive disorders and occur in persistent organic pollutants (POPs). Part of the debate over homosexuality involves whether taxpayers should be required to pay for others' Gender Reassignment surgeries.

Cancer Risk if Mixing Hormones
When women take estrogen, the female hormone, their risk of breast cancer declines. However, if taking more than just estrogen, their risk of breast cancer increased substantially, but declined once they stopped using artificial hormones.

"Women who took estrogen alone had a lower risk of breast cancer than women who took placebo. After nearly 11 years of follow-up, the risk of breast cancer among women who took estrogen alone remained lower than that among women who took placebo. Women who took estrogen plus progestin were more likely to be diagnosed with breast cancer than women who took placebo. The breast cancers in these women were larger and more likely to have spread to the lymph nodes by the time they were diagnosed. The risk of breast cancer was greater the longer women took the combined hormone therapy, but it decreased markedly when hormone use stopped." -National Institutes of Health, National Cancer Institute